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Plenary Symposium 3: Report on plenary symposium – The role of inflammation in “non-inflammatory” neurological diseases,
This plenary symposium captured the overarching theme of the Congress. Professor Vezzani from Milan began by discussing neuroinflammatory pathways as treatment targets and biomarker candidates in epilepsy. There is evidence for oxidative stress and neuroinflammatory responses in various forms of epilepsy, from animal models and surgical resection specimens, with microglial activation, extravasation of albumin, and upregulation of proinflammatory cytokines, such as interleukin 1-beta.