A very interesting paper recently published on PNAS sheds light on the mechanisms that connect social stress to increased risk of disease. Authors found that such effects are possible trough the “wires” of the sympathetic nervous system which up-regulate bone marrow production of immature, proinflammatory monocytes. In particular, these effects are mediated by β-adrenergic receptors opening up for new possible drug interventions to protect health in the context of chronic social stress. In the Neuroscience blog you can find an exhaustive summary of the paper.
Enjoy the reading and…relax!
Delia Lenzi, M.D., Ph. D.
